Stress
Selected abstracts:
1.
Bax JJ,
Bonow RO,
Tschope D,
Inzucchi SE,
Barrett E;
Global Dialogue Group
for the Evaluation of Cardiovascular Risk in Patients With Diabetes.
The potential of myocardial perfusion scintigraphy for risk stratification
of asymptomatic patients with type 2 diabetes.
J Am Coll Cardiol.
2006 Aug 15;48(4):754-60.
Leiden University Medical Center, Leiden, The
Netherlands.
jbax@knoware.nl
Patients with diabetes, in particular
patients with type 2 diabetes, are at a 2- to 4-fold higher risk of
cardiovascular mortality compared with their nondiabetic peers. Patients
with diabetes are also more likely to have silent ischemia and less likely
to survive a myocardial infarction than nondiabetic patients. Recent studies
with electron beam computed tomography (EBCT) have shown that subclinical
atherosclerosis is common in patients with diabetes, and studies with
myocardial perfusion scintigraphy (with single-photon emission computed
tomography) or stress echocardiography have demonstrated that between 25%
and 50% of asymptomatic diabetic patients have ischemia during exercise or
pharmacological stress and that a substantial proportion of these patients
go on to develop major cardiovascular events within several years. Clearly,
asymptomatic diabetic patients include a subset of individuals at high risk
of cardiovascular disease who would benefit from improved risk
stratification beyond that possible with risk factor scoring systems alone.
Single-photon emission computed tomography, stress echocardiography, and
possibly EBCT or multi-slice computed tomography, are emerging as valuable
diagnostic tools for identifying asymptomatic diabetic patients who might
require early and aggressive intervention to manage their cardiovascular
risk.
1.
Breslau N. Neurobiological research on sleep and stress hormones in
epidemiological samples. Ann N Y Acad Sci. 2006 Jul;1071:221-30. Review.
Department of Epidemiology, Michigan State University,
B645 West Fee Hall, East Lansing, MI 48824, USA.
Information on stress hormones and sleep disturbance in
posttraumatic stress disorder (PTSD) is on the basis of clinical samples and
samples of other selective populations. Neurobiological studies nested in a
large epidemiological community sample were recently reported. PTSD was
compared with several control groups, defined by exposure and by diagnostic
classification on the basis of comorbidity with Major Depression. Key
findings were: (a) higher mean catecholamines in persons with PTSD versus
controls; (b) no difference in mean cortisol between groups; (c) comorbid
PTSD and depression was associated with higher cortisol in women; and (d)
polysomnographic studies failed to detect clinically relevant sleep
disturbance in PTSD, although an increase in brief arousal from REM was
detected. Methodological questions raised by discrepancies between
biological findings from epidemiologic versus clinical and other selective
samples are discussed.
2.
Haidara MA, Yassin HZ, Rateb M, Ammar H,
Zorkani MA. Role of oxidative stress in
development of cardiovascular complications in diabetes mellitus.
Curr Vasc Pharmacol. 2006 Jul;4(3):215-27.
Review.
Department of Physiology, Kasr Al-Aini Faculty of Medicine, Cairo
University, Egypt. haidaram@hotmail.com
Diabetes represents a serious risk factor for the
development of cardiovascular problems such as coronary heart disease,
peripheral arterial disease, hypertension, stroke, cardiomyopathy,
nephropathy and retinopathy. Identifying the pathogenesis of this increased
risk provides a basis for secondary intervention to reduce morbidity and
mortality in diabetic patients. Hyperglycemia and protein glycation,
increased inflammation, a prothrombotic state and endothelial dysfunction
have all been implicated as possible mechanisms for such complications. A
linking element between many of these phenomena could possibly be, among
other factors, increased production of reactive oxygen species. Vascular
endothelial cells have several physiological actions that are essential for
the normal function of the cardiovascular system. These include the
production of nitric oxide (NO), which regulates vasodilatation,
anticoagulation, leukocyte adhesion, smooth muscle proliferation and the
antioxidative capacity of endothelial cells. However, under conditions of
hyperglycemia, excessive amounts of superoxide radicals are produced inside
vascular cells and this can interfere with NO production leading to the
possible complications. This article aims at reviewing the links between
reactive oxygen species, diabetes and vascular disease and whether or not
antioxidants can alter the course of vascular complications in diabetic
patients and animal models. A possible beneficial effect of antioxidants
might present a new addition to the range of secondary preventive measures
used in diabetic patients.
4.
Harmsen P, Lappas G, Rosengren A, Wilhelmsen L. Long-term risk
factors for stroke: twenty-eight years of follow-up of 7457 middle-aged men
in Goteborg, Sweden. Stroke. 2006 Jul;37(7):1663-7 .
Institute of Clinical Neuroscience, Sahlgrenska University Hospital,
Goteborg, Sweden.
BACKGROUND AND PURPOSE: To estimate the predictive value of risk factors for
stroke measured in midlife over follow-up extending through 28 years.
METHODS: A cohort of 7457 men 47 to 55 years of age and free of stroke at
baseline year 1970 were examined. Risk of stroke was analyzed for the entire
period and for 0 to 15, 16 to 21, and 22 to 28 years of follow-up using
age-adjusted and multiple Cox regression analyses. RESULTS: Age, diabetes,
and high blood pressure were independently associated with increased risk of
stroke for the entire 28 years and for each of the periods. Previous
transient ischemic attacks, atrial fibrillation, history of chest pain,
smoking, and psychological stress were independently related to stroke for
the entire follow-up period and also during the first 1 or 2 successive
periods. Family history of stroke or of coronary disease carried no
independent prognostic information, nor did serum cholesterol. Elevated body
mass index predicted stroke during the later part of the follow-up and so
did (almost) low physical activity during leisure time, together with
antihypertensive medication at baseline. CONCLUSIONS: High blood pressure
and diabetes retain their importance as stroke risk factors also over an
extended follow-up into old age. A family history of cardiovascular disease
was not significantly related to outcome. Transient ischemic attacks, atrial
fibrillation, stress, smoking, and a history of chest pain were associated
with outcome only for the first or the first 2 periods. High body mass index
and antihypertensive medication at baseline emerged as risk factors in the
second and third decades.
5.
Levinson DF. The genetics of depression: a review. Biol Psychiatry.
2006 Jul 15;60(2):84-92.
Department of Psychiatry, Center for Neurobiology and
Behavior, University of Pennsylvania School of Medicine, Philadelphia,
Pennsylvania 19104-3309, USA.
Major depressive disorder (MDD) is common and moderately heritable.
Recurrence and early age at onset characterize cases with the greatest
familial risk. Major depressive disorder and the neuroticism personality
trait have overlapping genetic susceptibilities. Most genetic studies of MDD
have considered a small set of functional polymorphisms relevant to
monoaminergic neurotransmission. Meta-analyses suggest small positive
associations between the polymorphism in the serotonin transporter promoter
region (5-HTTLPR) and bipolar disorder, suicidal behavior, and
depression-related personality traits but not yet to MDD itself. This
polymorphism might also influence traits related to stress vulnerability.
Newer hypotheses of depression neurobiology suggest closer study of genes
related to neurotoxic and neuroprotective (neurotrophic) processes and to
overactivation of the hypothalamic-pituitary axis, with mixed evidence
regarding association of MDD with polymorphisms in one such gene
(brain-derived neurotrophic factor [BDNF]). Several genome-wide linkage
studies of MDD and related traits have been reported or are near completion.
There is some evidence for convergence of linkage findings across studies,
but more data are needed to permit meta-analysis. Future directions will
include more intensive, systematic study of linkage candidate regions and of
the whole genome for genetic association; gene expression array studies; and
larger-scale studies of gene-environment interactions and of
depression-related endophenotypes.
6.
Olson AL, Dietrich AJ, Prazar G, Hurley J. Brief maternal depression
screening at well-child visits. Pediatrics. 2006 Jul;118(1):207-16.
Department of Pediatrics, Dartmouth Hitchcock Medical Center, HB
7450, 1 Medical Center Dr, Lebanon, New Hampshire 03756-0011, USA
OBJECTIVES: The goals were (1) to determine the feasibility and yield of
maternal depression screening during all well-child visits, (2) to
understand how pediatricians and mothers respond to depression screening
information, and (3) to assess the time required for discussion of screening
results. METHODS: Implementation of brief depression screening of mothers at
well-child visits for children of all ages was studied in 3 rural pediatric
practices. Two screening trials introduced screening (1 month) and then
determined whether screening could be sustained (6 months). Screening used
the 2-question Patient Health Questionnaire. Practices tracked the
proportions of visits screened and provided data about the screening
process. RESULTS: Practices were able to screen in the majority of
well-child visits (74% in trial 1 and 67% in trial 2). Of 1398 mothers
screened, 17% had 1 of the depressive symptoms and 6% (n = 88) scored as
being at risk for a major depressive disorder. During discussion, 5.7% of
all mothers thought they might be depressed and 4.7% thought they were
stressed but not depressed. Pediatric clinicians intervened with 62.4% of
mothers who screened positive and 38.2% of mothers with lesser symptoms.
Pediatrician actions included discussion of the impact on the child, a
follow-up visit or call, and referral to an adult primary care provider, a
mental health clinician, or community supports. Pediatrician time needed to
discuss screening results decreased in the second trial. Prolonged
discussion time was uncommon (5-10 minutes in 3% of all well-child visits
and >10 minutes in 2%). CONCLUSIONS: Routine, brief, maternal depression
screening conducted during well-child visits was feasible and detected
mothers who were willing to discuss depression and stress issues with their
pediatrician. The discussion after screening revealed additional mothers who
felt depressed among those with lesser symptoms. The additional discussion
time was usually brief and resulted in specific pediatrician actions.
7.
Ross LE, McLean LM. Anxiety disorders during pregnancy and the
postpartum period: A systematic review. J Clin Psychiatry. 2006
Aug;67(8):1285-98. Review.
Women's Mental Health and Addiction Research Section, Centre for Addiction
and Mental Health, Department of Psychiatry, University of Toronto, Toronto,
Ontario, Canada.
OBJECTIVE: The postpartum period is recognized as a time of vulnerability to
affective disorders, particularly postpartum depression. In contrast, the
prevalence and clinical presentation of anxiety disorders during pregnancy
and the postpartum period have received little research attention. In this
article, we review the medical literature as it relates to the prevalence
and clinical presentation of panic disorder, obsessive-compulsive disorder,
posttraumatic stress disorder, and generalized anxiety disorder during
pregnancy and the postpartum period. DATA SOURCES: MEDLINE (1966 to July
2005 week 1) and PsycInfo (1840 to July 2005 week 1) were searched using
combinations of the following search terms: pregnancy, childbirth,
postpartum, panic disorder, phobia, obsessive-compulsive disorder,
posttraumatic stress disorder, and generalized anxiety disorder. STUDY
SELECTION: All relevant papers published in English and reporting original
data related to perinatal anxiety disorders were included. DATA EXTRACTION:
Studies were examined for data related to the prevalence, presentation,
predictors/risk factors, new onset, course, and treatment of anxiety
disorders during pregnancy and the postpartum period. DATA SYNTHESIS:
Anxiety disorders are common during the perinatal period, with reported
rates of obsessive-compulsive disorder and generalized anxiety disorder
being higher in postpartum women than in the general population. The
perinatal context of anxiety disorders presents unique issues for detection
and management. CONCLUSIONS: Future research is needed to estimate the
prevalence of perinatal anxiety disorders more precisely, to identify
potential implications of maternal anxiety disorders for maternal quality of
life and child development, and to determine safe and effective treatment
methods.
8.
Simon NM, Smoller JW, McNamara KL, Maser RS, Zalta AK, Pollack MH,
Nierenberg AA, Fava M, Wong KK. Telomere shortening and mood disorders:
preliminary support for a chronic stress model of accelerated aging. Biol
Psychiatry. 2006 Sep 1;60(5):432-5.
Department of Psychiatry, Massachusetts General Hospital and Harvard Medical
School, Boston, MA 02114, USA. NSIMON@Partners.org
BACKGROUND: Little is known about the biological mechanisms underlying the
excess medical morbidity and mortality associated with mood disorders.
Substantial evidence supports abnormalities in stress-related biological
systems in depression. Accelerated telomere shortening may reflect
stress-related oxidative damage to cells and accelerated aging, and severe
psychosocial stress has been linked to telomere shortening. We propose that
chronic stress associated with mood disorders may contribute to excess
vulnerability for diseases of aging such as cardiovascular disease and
possibly some cancers through accelerated organismal aging. METHODS:
Telomere length was measured by Southern Analysis in 44 individuals with
chronic mood disorders and 44 nonpsychiatrically ill age-matched control
subjects. RESULTS: Telomere length was significantly shorter in those with
mood disorders, representing as much as 10 years of accelerated aging.
CONCLUSIONS: These results provide preliminary evidence that mood disorders
are associated with accelerated aging and may suggest a novel mechanism for
mood disorder-associated morbidity and mortality.
9.
Thompson JB, Rivera JJ, Blumenthal RS, Danyi P. Primary prevention
for patients with intermediate Framingham risk scores. Curr Cardiol Rep.
2006 Jul;8(4):261-6. Review.
Coronary heart disease (CHD) is the leading cause of
mortality in the industrialized world. Proper identification of individuals
at risk for CHD is challenging. The Framingham Risk Score, the most widely
accepted tool for quantifying 10-year risk, fails to identify a great
proportion of future CHD. Because of the health and economic consequences of
CHD, there is a need to develop better prognostic tools for primary
prevention. Coronary artery calcium scoring, C-reactive protein measurement,
and heart rate recovery and exercise tolerance during exercise stress test
may be useful tools for better risk stratification of intermediate-risk
patients.
10.
Wolber T, Maeder M, Weilenmann D, Duru F, Bluzaite I, Riesen W,
Rickli H, Ammann P. Integration of B-type natriuretic peptide levels with
clinical data and exercise testing for predicting coronary artery disease.
Am J Cardiol. 2006 Sep 15;98(6):764-7
Cardiovascular Center, Cardiology, University Hospital Zurich, Zurich,
Switzerland.
thomas.wolber@usz.ch
Natriuretic peptides have been shown to be high in patients with myocardial
ischemia. We sought to create a diagnostic score using clinical data, stress
testing, and B-type natriuretic peptide (BNP) levels to improve noninvasive
prediction of coronary artery disease (CAD). Patients with stable angina
pectoris and normal systolic left ventricular function were eligible for
this prospective cohort study. Patients with arrhythmias, valvular heart
disease, impaired left ventricular function, or renal dysfunction were
excluded. All patients underwent clinical evaluation, bicycle stress
testing, BNP testing, and coronary angiography. Then a diagnostic risk score
was derived that combined cardiovascular risk factors, results of exercise
testing, and BNP measurements and added 1 point for the presence of each of
these variables. Seventy-one patients (52 years of age, range 31 to 61; 46
men) were included in the study. Prevalence of CAD, defined by 50% narrowing
of > or =1 coronary artery on coronary angiography, was 45%. For 0 point in
the risk score system, the negative predictive value was 93% with a negative
likelihood ratio of 0.1 (95% confidence interval [CI] 0.02 to 0.38); for a
score of 3 points, the positive predictive value was 93% with a positive
likelihood ratio of 15.9 (95% CI 2.19 to 114.7). Serum BNP level >50 ng/L at
rest was the best single diagnostic parameter, with 66% sensitivity and 97%
specificity, and a positive likelihood ratio of 25.6 (95% CI 3.64 to 180)
and a negative likelihood ratio of 0.35 (95% CI 0.22 to 0.57). In
conclusion, a diagnostic score combining exercise testing, clinical data,
and serum BNP values at rest can distinguish patients with CAD from those
without CAD with high accuracy.
Asthma:
15141.
Ippoliti F, De Santis W, Volterrani A, Canitano N, Frattolillo D,
Lucarelli S, Frediani S, Frediani T. Psychological stress affects response
to sublingual immunotherapy in asthmatic children allergic to house dust
mite. Pediatr Allergy Immunol. 2006 Aug;17(5):337-45.
15142.
Pohlman B, Becker G. "Stress knocks hard on your immune system":
asthma and the discourse on stress. Med Anthropol. 2006
Jul-Sep;25(3):265-95.
15143.
Saposnik G, Baibergenova A, Dang J, Hachinski V. Does a birthday
predispose to vascular events? Neurology. 2006 Jul 25;67(2):300-4.
15144.
Shimizu Y, Dobashi K, Kobayashi S, Ohki I, Tokushima M, Kusano M,
Kawamura O, Shimoyama Y, Utsugi M, Sunaga N, Ishizuka T, Mori M. A proton
pump inhibitor, lansoprazole, ameliorates asthma symptoms in asthmatic
patients with gastroesophageal reflux disease.
Tohoku J Exp Med. 2006 Jul;209(3):181-9.
Depression:
15145.
Amirkhanyan AA, Wolf DA. Parent care and the stress process: findings
from panel data. J Gerontol B Psychol Sci Soc Sci. 2006 Sep;61(5):S248-55.
15146.
Ando T, Hashiro M, Noda K, Adachi J, Hosoya R, Kamide R, Ishikawa T,
Komaki G. Development and validation of the psychosomatic scale for atopic
dermatitis in adults. J Dermatol. 2006
Jul;33(7):439-50.
15147.
Angermeyer MC, Bull N, Bernert S, Dietrich S, Kopf A. Burnout of
caregivers: a comparison between partners of psychiatric patients and
nurses. Arch Psychiatr Nurs. 2006 Aug;20(4):158-65.
15148.
Arving C, Sjoden PO, Bergh J, Lindstrom AT,
Wasteson E, Glimelius B, Brandberg Y. Satisfaction, utilisation and
perceived benefit of individual psychosocial support for breast cancer
patients--a randomised study of nurse versus psychologist interventions.
Patient Educ Couns. 2006 Aug;62(2):235-43.
15149.
Brown ES, Beard L, Frol AB, Rush AJ. Effect of two prednisone
exposures on mood and declarative memory. Neurobiol Learn Mem. 2006
Jul;86(1):28-34.
15150.
Campagne DM. Should fertilization treatment start with reducing
stress? Hum Reprod. 2006 Jul;21(7):1651-8.
15151.
Dyl J, Kittler J, Phillips KA, Hunt JI. Body dysmorphic disorder and
other clinically significant body image concerns in adolescent psychiatric
inpatients: prevalence and clinical characteristics. Child Psychiatry Hum
Dev. 2006 Summer;36(4):369-82.
15152.
Evans-Campbell T, Lindhorst T, Huang B, Walters KL. Interpersonal
violence in the lives of urban American Indian and Alaska Native women:
implications for health, mental health, and help-seeking. Am J Public
Health. 2006 Aug;96(8):1416-22.
15153.
Fulkerson JA, Story M, Mellin A, Leffert N, Neumark-Sztainer D,
French SA. Family dinner meal frequency and adolescent development:
relationships with developmental assets and high-risk behaviors. J Adolesc
Health. 2006 Sep;39(3):337-45.
15154.
Gibson EL. Emotional influences on food
choice: sensory, physiological and psychological pathways. Physiol Behav.
2006 Aug 30;89(1):53-61.
15155.
Goubert L, Eccleston C, Vervoort T, Jordan
A, Crombez G. Parental catastrophizing about their child's pain. The parent
version of the Pain Catastrophizing Scale (PCS-P): a preliminary validation.
Pain. 2006 Aug;123(3):254-63.
15156.
Hauger RL, Risbrough V, Brauns O, Dautzenberg FM. Corticotropin
releasing factor (CRF) receptor signaling in the central nervous system: new
molecular targets. CNS Neurol Disord Drug Targets. 2006 Aug;5(4):453-79.
Review.
15157.
Herringa RJ, Roseboom PH, Kalin NH. Decreased amygdala CRF-binding
protein mRNA in post-mortem tissue from male but not female bipolar and
schizophrenic subjects. Neuropsychopharmacology. 2006 Aug;31(8):1822-31.
15158.
Houry D, Kemball R, Rhodes KV, Kaslow NJ. Intimate partner violence
and mental health symptoms in African American female ED patients. Am J
Emerg Med. 2006 Jul;24(4):444-50.
15159.
Inslicht SS, Marmar CR, Neylan TC, Metzler TJ, Hart SL, Otte C,
McCaslin SE, Larkin GL, Hyman KB, Baum A. Increased cortisol in women with
intimate partner violence-related posttraumatic stress disorder.
Psychoneuroendocrinology. 2006 Aug;31(7):825-38.
15160.
Jelinek L, Jacobsen D, Kellner M, Larbig F, Biesold KH, Barre K,
Moritz S. Verbal and nonverbal memory functioning in posttraumatic stress
disorder (PTSD). J Clin Exp Neuropsychol. 2006 Aug;28(6):940-8.
15161.
Johannsen A, Rylander G, Soder B, Asberg M. Dental plaque, gingival
inflammation, and elevated levels of interleukin-6 and cortisol in gingival
crevicular fluid from women with stress-related depression and exhaustion. J
Periodontol. 2006 Aug;77(8):1403-9.
15162.
Kinzie JD, Cheng K, Tsai J, Riley C.
Traumatized refugee children: the case for individualized diagnosis and
treatment. J Nerv Ment Dis. 2006
Jul;194(7):534-7.
15163.
Kissane DW, McKenzie M, Bloch S, Moskowitz
C, McKenzie DP, O'Neill I. Family focused grief
therapy: a randomized, controlled trial in palliative care and bereavement.
Am J Psychiatry. 2006 Jul;163(7):1208-18.
15164.
Kitamura T, Hasui C. Anger feelings and
anger expression as a mediator of the effects of witnessing family violence
on anxiety and depression in Japanese adolescents.
J Interpers Violence. 2006 Jul;21(7):843-55.
15165.
Lathers CM, Schraeder PL. Stress and sudden death. Epilepsy Behav.
2006 Sep;9(2):236-42.
15166.
Marcellini F, Giuli C, Papa R, Malavolta M, Mocchegiani E.
Psychosocial aspects and zinc status: is there a relationship with
successful aging? Rejuvenation Res. 2006 Summer;9(2):333-7.
15167.
Mazzaferro KE, Murray PJ, Ness RB, Bass DC, Tyus N, Cook RL.
Depression, stress, and social support as predictors of high-risk sexual
behaviors and STIs in young women. J Adolesc Health. 2006 Oct;39(4):601-3.
15168.
McCallum TJ, Sorocco KH, Fritsch T. Mental
health and diurnal salivary cortisol patterns among African American and
European American female dementia family caregivers. Am J Geriatr
Psychiatry. 2006 Aug;14(8):684-93.
15169.
Miles R, Cowan F, Glover V, Stevenson J, Modi N. A controlled trial
of skin-to-skin contact in extremely preterm infants. Early Hum Dev. 2006
Jul;82(7):447-55.
15170.
Orr G, Weiser M, Polliack M, Raviv G,
Tadmor D, Grunhaus L. Effectiveness of sildenafil
in treating erectile dysfunction in PTSD patients: a double-blind,
placebo-controlled crossover study. J Clin Psychopharmacol. 2006
Aug;26(4):426-30.
15171.
Pence BW, Miller WC, Whetten K, Eron JJ,
Gaynes BN. Prevalence of DSM-IV-defined mood, anxiety, and substance use
disorders in an HIV clinic in the Southeastern United States. J Acquir
Immune Defic Syndr. 2006 Jul;42(3):298-306.
15172.
Recklitis CJ, Lockwood RA, Rothwell MA, Diller LR. Suicidal ideation
and attempts in adult survivors of childhood cancer. J Clin Oncol. 2006 Aug
20;24(24):3852-7.
15173.
Segal ZV, Kennedy S, Gemar M, Hood K, Pedersen
R, Buis T. Cognitive reactivity to sad mood
provocation and the prediction of depressive relapse. Arch Gen Psychiatry.
2006 Jul;63(7):749-55.
15174.
Stewart DW, de Vries J, Singer DL, Degen GG, Wener P. Canadian dental
students' perceptions of their learning environment and psychological
functioning over time. J Dent Educ. 2006 Sep;70(9):972-81.
15175.
Ter Bogt TF, van Dorsselaer SA, Monshouwer
K, Verdurmen JE, Engels RC, Vollebergh WA. Body
mass index and body weight perception as risk factors for internalizing and
externalizing problem behavior among adolescents. J Adolesc Health. 2006
Jul;39(1):27-34.
15176.
Waldinger MD, Schweitzer DH. Changing paradigms from a historical
DSM-III and DSM-IV view toward an evidence-based definition of premature
ejaculation. Part I--validity of DSM-IV-TR. J Sex Med. 2006 Jul;3(4):682-92.
Review.
15177.
Whitaker RC, Phillips SM, Orzol SM. Food insecurity and the risks of
depression and anxiety in mothers and behavior problems in their
preschool-aged children. Pediatrics. 2006 Sep;118(3):e859-68.
Hypertension:
15178.
Ariff B, Zambanini A, Vamadeva S, Barratt D, Xu Y, Sever P, Stanton
A, Hughes A, Thom S. Candesartan- and atenolol-based treatments induce
different patterns of carotid artery and left ventricular remodeling in
hypertension. Stroke. 2006 Sep;37(9):2381-4.
15179.
Finsterer J, Gelpi E. Mitochondrial disorder aggravated by
propranolol. South Med J. 2006 Jul;99(7):768-71.
15180.
Konukoglu D, Serin O, Turhan MS.
Plasma leptin and its relationship with lipid
peroxidation and nitric oxide in obese female patients with or without
hypertension. Arch Med Res. 2006 Jul;37(5):602-6.
15181.
Moreno MU, Jose GS, Fortuno A, Beloqui O, Diez J, Zalba G. The C242T
CYBA polymorphism of NADPH oxidase is associated with essential
hypertension. J Hypertens. 2006 Jul;24(7):1299-306.
15182.
Palatini P, Longo D, Zaetta V, Perkovic D,
Garbelotto R, Pessina AC. Evolution of blood
pressure and cholesterol in stage 1 hypertension: role of autonomic nervous
system activity. J Hypertens. 2006
Jul;24(7):1375-81.
15183.
Sukhija R, Kakar P, Mehta V, Mehta JL. Enhanced 11beta-hydroxysteroid
dehydrogenase activity, the metabolic syndrome, and systemic hypertension.
Am J Cardiol. 2006 Aug 15;98(4):544-8.
15184.
Taddei S, Versari D, Cipriano A, Ghiadoni L, Galetta F, Franzoni F,
Magagna A, Virdis A, Salvetti A. Identification of a cytochrome P450
2C9-derived endothelium-derived hyperpolarizing factor in essential
hypertensive patients. J Am Coll Cardiol. 2006 Aug 1;48(3):508-15.
15185.
Thomaides T, Karapanayiotides T, Spantideas A, Andrikou A. Are
transient increases in blood pressure during the treadmill stress test
associated with headache? Cephalalgia. 2006 Jul;26(7):837-42.
15186.
Xu S, Touyz RM. Reactive oxygen species and vascular remodelling in
hypertension: still alive. Can J Cardiol. 2006 Sep;22(11):947-51. Review.
15187.
Yang H, Schnall PL, Jauregui M, Su TC, Baker D. Work hours and
self-reported hypertension among working people in California. Hypertension.
2006 Oct;48(4):744-50.
Heart Disease:
15188.
Abe N, Matsunaga T, Kameda K, Tomita H, Fujiwara T, Ishizaka H,
Hanada H, Fukui K, Fukuda I, Osanai T, Okumura K. Increased level of
pericardial insulin-like growth factor-1 in patients with left ventricular
dysfunction and advanced heart failure. J Am Coll Cardiol. 2006 Oct
3;48(7):1387-95.
15189.
Adamopoulos S, Parissis JT, Iliodromitis EK,
Paraskevaidis I, Tsiapras D, Farmakis D, Karatzas D, Gheorghiade M,
Filippatos GS, Kremastinos DT. Effects of levosimendan versus dobutamine
on inflammatory and apoptotic pathways in acutely decompensated chronic
heart failure. Am J Cardiol. 2006 Jul 1;98(1):102-6.
15190.
Bella JN, Palmieri V, Roman MJ, Paranicas
MF, Welty TK, Lee ET, Fabsitz RR, Howard BV, Devereux RB.
Gender differences in left ventricular systolic function
in American Indians (from the Strong Heart Study). Am J Cardiol. 2006 Sep
15;98(6):834-7.
15191.
Corrrigan M, Cupples ME, Smith SM, Byrne M, Leathem CS, Clerkin P,
Murphy AW. The contribution of qualitative research in designing a complex
intervention for secondary prevention of coronary heart disease in two
different healthcare systems. BMC Health Serv Res. 2006 Jul 18;6:90.
15192.
Edelman D, Oddone EZ, Liebowitz RS, Yancy WS Jr, Olsen MK, Jeffreys
AS, Moon SD, Harris AC, Smith LL, Quillian-Wolever RE, Gaudet TW. A
multidimensional integrative medicine intervention to improve cardiovascular
risk. J Gen Intern Med. 2006 Jul;21(7):728-34.
15193.
Giallauria F, Lucci R, Pietrosante M, Gargiulo G, De Lorenzo A,
D'Agostino M, Gerundo G, Abete P, Rengo F, Vigorito C. Exercise-based
cardiac rehabilitation improves heart rate recovery in elderly patients
after acute myocardial infarction. J Gerontol A Biol Sci Med Sci. 2006
Jul;61(7):713-7.
15194.
Haidara MA, Yassin HZ, Rateb M, Ammar H, Zorkani MA. of oxidative
stress in development of cardiovascular complications in diabetes mellitus.
Curr Vasc Pharmacol. 2006 Jul;4(3):215-27.
Review.
15195.
Hankey GJ. Potential new risk factors for ischemic stroke: what is
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